Results for MAPK9

General Information

Gene ID 5601
Gene Symbol MAPK9
Gene Name mitogen-activated protein kinase 9
Gene Type protein-coding
Cytoband 5q35
Ensembl ID ENSG00000050748
#miR regulators 23
Omim ID 602896
Gene ontology GO:0001836: release of cytochrome c from mitochondria
GO:0001934: positive regulation of protein phosphorylation
GO:0002224: toll-like receptor signaling pathway
GO:0002755: MyD88-dependent toll-like receptor signaling pathway
GO:0002756: MyD88-independent toll-like receptor signaling pathway
GO:0006626: protein targeting to mitochondrion
GO:0009612: response to mechanical stimulus
GO:0006950: response to stress
GO:0007254: JNK cascade
GO:0007417: central nervous system development
GO:0042493: response to drug
GO:0009636: response to toxic substance
GO:0010628: positive regulation of gene expression
GO:0010744: positive regulation of macrophage derived foam cell differentiation
GO:0010770: positive regulation of cell morphogenesis involved in differentiation
GO:0014075: response to amine stimulus
GO:0045893: positive regulation of transcription, DNA-dependent
GO:0046686: response to cadmium ion
GO:0031175: neuron projection development
GO:0031394: positive regulation of prostaglandin biosynthetic process
GO:0031396: regulation of protein ubiquitination
GO:0032308: positive regulation of prostaglandin secretion
GO:0032722: positive regulation of chemokine production
GO:0034134: toll-like receptor 2 signaling pathway
GO:0034138: toll-like receptor 3 signaling pathway
GO:0034142: toll-like receptor 4 signaling pathway
GO:0034146: toll-like receptor 5 signaling pathway
GO:0034162: toll-like receptor 9 signaling pathway
GO:0034166: toll-like receptor 10 signaling pathway
GO:0034644: cellular response to UV
GO:0035666: TRIF-dependent toll-like receptor signaling pathway
GO:0038095: Fc-epsilon receptor signaling pathway
GO:0038123: toll-like receptor TLR1:TLR2 signaling pathway
GO:0038124: toll-like receptor TLR6:TLR2 signaling pathway
GO:0043065: positive regulation of apoptotic process
GO:0045087: innate immune response
GO:0045429: positive regulation of nitric oxide biosynthetic process
GO:0046328: regulation of JNK cascade
GO:0051090: regulation of sequence-specific DNA binding transcription factor activity
GO:0051403: stress-activated MAPK cascade
GO:0051770: positive regulation of nitric-oxide synthase biosynthetic process
GO:0071222: cellular response to lipopolysaccharide
GO:0071347: cellular response to interleukin-1
GO:0071356: cellular response to tumor necrosis factor
GO:0071363: cellular response to growth factor stimulus
GO:2001235: positive regulation of apoptotic signaling pathway
GO:0005739: mitochondrion
GO:0005829: cytosol
GO:0005654: nucleoplasm
GO:0005515: protein binding
GO:0004705: JUN kinase activity
GO:0005524: ATP binding
GO:0008134: transcription factor binding
GO:0008656: cysteine-type endopeptidase activator activity involved in apoptotic process
GO:0031435: mitogen-activated protein kinase kinase kinase binding
KEGG pathways 4010: MAPK signaling pathway
4012: ErbB signaling pathway
4141: Protein processing in endoplasmic reticulum
4310: Wnt signaling pathway
4380: Osteoclast differentiation
4510: Focal adhesion
4620: Toll-like receptor signaling pathway
4621: NOD-like receptor signaling pathway
4622: RIG-I-like receptor signaling pathway
4660: T cell receptor signaling pathway
4664: Fc epsilon RI signaling pathway
4722: Neurotrophin signaling pathway
4910: Insulin signaling pathway
4912: GnRH signaling pathway
4914: Progesterone-mediated oocyte maturation
4920: Adipocytokine signaling pathway
4930: Type II diabetes mellitus
5120: Epithelial cell signaling in Helicobacter pylori infection
5131: Shigellosis
5142: Chagas disease (American trypanosomiasis)
5145: Toxoplasmosis
5160: Hepatitis C
5200: Pathways in cancer
5210: Colorectal cancer
5212: Pancreatic cancer

PubMed abstracts associated with MAPK9

PMID Title Tumor Value
12689679 Activation of the JNK-AP-1 signal transduction pathway is associated with pathogenesis and progression of human osteosarcomas. yes yes
title all all